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C., Xia W., Chen I. cells activated appearance of activating transcription aspect 4 (ATF4), a get good at transcription aspect for stress-induced apoptosis, through activation of its target proapoptotic genes and RET knockdown increased sensitivity to cisplatin-induced apoptosis also. We observed that RET interacted with and phosphorylated ATF4 at tyrosine and threonine residues physically. Certainly, …